Which of the following is NOT an effect of Angiotensin II in the RAAS?

Angiotensin II is a key component of the Renin-Angiotensin-Aldosterone System (RAAS) and is primarily known for its role in regulating blood pressure and fluid balance. It has several physiological effects that contribute to these regulatory processes.

One significant effect of Angiotensin II is its ability to increase the secretion of aldosterone from the adrenal cortex. Aldosterone promotes sodium reabsorption in the kidneys, which in turn helps to increase water retention, thus raising blood volume and blood pressure. Similarly, Angiotensin II stimulates the release of antidiuretic hormone (ADH), which enhances water reabsorption in the kidneys, further aiding in fluid retention and blood pressure control.

Angiotensin II also directly increases sodium and water reabsorption by acting on various regions of the nephron, particularly in the proximal tubules. These actions contribute to the overall increase in blood volume and systemic vascular resistance, thereby elevating blood pressure.

In contrast, the statement regarding the induction of vasodilation is incorrect. Angiotensin II primarily causes vasoconstriction, which raises blood pressure by increasing peripheral resistance. Therefore, it does not induce vasodilation, making this the answer that does not align with the known effects of